Dyslexia is considered to be a learning disability. It manifests primarily as a difficulty with written language, particularly with reading and spelling. It is separate and distinct from reading difficulties resulting from other causes, such as a non-neurological deficiency with vision or hearing, or from poor or inadequate reading instruction. Evidence also suggests that dyslexia results from differences in how the brain processes written and/or spoken language. Although dyslexia is allegedly the result of a neurological difference, it is not an intellectual disability. Dyslexia has been diagnosed in people possessing all levels of intelligence.
The word dyslexia comes from the Greek words δυσ- dys- ("impaired") and λέξις lexis ("word").
Dyslexia is most commonly characterized by difficulties with learning how to decode words, to spell, and to read accurately and fluently. Dyslexia can also lead to difficulties in mathematics due to challenges in comprehension and sequencing. Dyslexic individuals often have difficulty "breaking the code" of sound-letter association (the alphabetic principle), and they may also reverse or transpose letters when writing or confuse letters such as b, d, p, q, especially in childhood. However, dyslexia is not a visual problem that involves reading letters or words backwards or upside down, nor are such reversals a defining characteristic of dyslexia.
Many individuals with dyslexic symptoms involving reading, writing, and spelling also exhibit symptoms in other domains such as poor short-term memory skills, poor personal organizational skills and problems processing spoken language. These symptoms may coexist with or overlap with characteristics among others of attention-deficit hyperactivity disorder, auditory processing disorder, developmental dyspraxia, dyscalculia, and/or dysgraphia. However, dyslexia and attention deficit/hyperactivity disorder are not correlated developmental problems.
Evidence that dyslexia is of neurological origin is substantial. Acquired dyslexias occur due to brain damage in the left hemisphere's key language areas, which may be a clue when it comes to developmental types. Research also suggests an association with biochemical and genetic markers. However, experts disagree over the precise definition and criteria for diagnosis, and some advocate that the term dyslexia be dropped altogether and replaced with the term reading disorder or reading disability (RD). Because reading skills occur on a continuum with no clear distinction between typical readers and dyslexic readers, some experts assert that the term dyslexia should be reserved for the two to five percent with the most severe reading deficits.
Dyslexia is a lifelong condition that cannot be reversed, but appropriate remedial instruction and compensatory strategies can help dyslexic individuals mitigate or overcome their difficulties with written language. A large body of evidence shows which types of instruction dyslexics need to be successful. Many dyslexics overcome early problems with literacy and go on to earn advanced degrees and pursue successful careers; a high level of motivation coupled with strong encouragement and mentorship have been identified as factors leading to their success.
In 1896, W. Pringle Morgan, a British physician, from Seaford, East Sussex, England published a description of a reading-specific learning disorder in a report to the British Medical Journal titled "Congenital Word Blindness". This described the case of a 14-year-old boy who had not yet learned to read, yet showed normal intelligence and was generally adept at other activities typical of children of that age.
During the 1890s and early 1900s, James Hinshelwood, a British ophthalmologist, published a series of articles in medical journals describing similar cases of congenital word blindness, which he defined as "a congenital defect occurring in children with otherwise normal and undamaged brains characterised by a difficulty in learning to read." In his 1917 book Congenital Word Blindness, Hinshelwood asserted that the primary disability was in visual memory for words and letters, and described symptoms including letter reversals, and difficulties with spelling and reading comprehension.
A key early researcher in dyslexia was Samuel T. Orton, a neurologist who worked primarily with stroke victims. In 1925 Orton met a boy who could not read and who exhibited symptoms similar to stroke victims who had lost the ability to read. Orton began studying reading difficulties and determined that there was a syndrome unrelated to brain damage that made learning to read difficult. Orton called the condition strephosymbolia (meaning 'twisted signs') to describe his theory that individuals with dyslexia had difficulty associating the visual forms of words with their spoken forms. Orton observed that reading deficits in dyslexia did not seem to stem from strictly visual deficits. He believed the condition was caused by the failure to establish hemispheric dominance in the brain. He also observed that the children he worked with were disproportionately left- or mixed-handed, although this finding has been difficult to replicate. Orton's hypothesis concerning hemispheric specialization was borne out by post-mortem studies in the 1980s and 1990s establishing that the left planum temporale, a brain area associated with language processing, is physically larger than the corresponding right area in the brains of non-dyslexic subjects, but that these brain areas are symmetrical or slightly larger on the right for dyslexic subjects. FMRI imaging studies of children and young adults reported in 2003 provide further support, demonstrating that increases in age and reading level are associated with a suppression of right hemispheric activity.
Influenced by the kinesthetic work of Helen Keller and Grace Fernald, and looking for a way to teach reading using both left and right brain functions, Orton later worked with psychologist and educator Anna Gillingham to develop an educational intervention that pioneered the use of simultaneous multisensory instruction. The Orton-Gillingham approach to remedial reading instruction is still widely used and forms the basis of many reading intervention programs.
In contrast, Dearborn, Gates, Bennet and Blau considered a faulty guidance of the seeing mechanism to be the cause. The data collected in 1931 by Tinker and Goodenough (The J.Educ. Psych.)(26) seemed to support this thesis. They wanted to know if a conflict between spontaneous orientation of the scanning action of the eyes from right to left and training aimed at the acquisition of an opposite direction would allow an interpretation of the facts observed in the dyslexic disorder and especially of the ability to mirror-read. To this end the authors asked four adults to read a text reflected in a mirror for ten minutes a day for a period of five months. It was confirmed that in all the subjects, the words were not perceived in their globality but required a meticulous analysis of the letters and syllables.
Moreover they also demonstrated either total or partial inversions even sometimes affecting the order of the words in a sentence. What is more, they revealed a curious impression of not just horizontal but also vertical inversions.These are errors that exist amongst dyslexics and they suffer from the aggravating circumstance inherent in all learning. What remained to be demonstrated was that there exists a preference amongst dyslexics, without sensory deficiency, or mental retardation, or any backwardness in speech or language, towards scanning with the eyes from right to left. Proof of this was provided in a work conducted under Clement Launay in 1949 (thesis G. Mahec Paris 1951). In adult subjects the reading of a series of 66 tiny lower-case letters, 5 mm high, spaced 5 mm apart, first from left to right and then from right to left was more easily and quickly done in the left to right direction. For former dyslexic children, a substantial number read a series of 42 letters with equal speed in both directions and some (10%) read better from right to left than from left to right. The phenomenon is clearly linked to the dynamics of sight as it disappears when the space between letters is increased, transforming the reading into spelling. This experience also explains the ability to mirror-read. This reading test can also be used to diagnose serious cases of dyslexia.
In the 1970s, a new hypothesis, based in part on Orton's theories, emerged that dyslexia stems from a deficit in phonological processing or difficulty in recognizing that spoken words are formed by discrete phonemes (for example, that the word CAT comes from the sounds [k], [æ], and [t]). As a result, affected individuals have difficulty associating these sounds with the visual letters that make up written words. Key studies of the phonological deficit hypothesis include the finding that the strongest predictor of reading success in school age children is phonological awareness, and that phonological awareness instruction can improve decoding skills for children with reading difficulties.
The advent of neuroimaging techniques to study brain structure and function enhanced the research in the 1980s and 1990s. Since then, interest in the neurologically based causes has persisted. Current models of the relation between the brain and dyslexia generally focus on some form of defective or delayed brain maturation. More recently, genetic research has provided increasing evidence supporting a genetic origin of dyslexia .
Researchers are currently searching for a link between the neurological and genetic findings, and the reading disorder. There are many previous and current theories of dyslexia, but the one that has the most support from research is that, whatever the biological cause, dyslexia is a matter of reduced phonogical awareness, the ability to analyze and link the units of spoken and written languages. .
Studies by Castles and Coltheart suggest that developmental dyslexia includes at least two prevalent and distinct varieties or subtypes of dyslexia. Subtypes include surface dyslexia and phonological dyslexia. Understanding these subtypes is useful in diagnosing learning patterns and developing approaches for overcoming impairments that may be visual perception impairments or speech discrimination deficits. These subtypes are based on differing patterns of underlying symptoms, as supported by a finding using large-scale data from comparative studies of reading patterns in dyslexic and normal readers . In the study by Castles and Coltheart, 56 dyslexic boys and 56 non-dyslexic boys as a control group were tested. During the test, the boys read aloud words and non words that were presented to them. The researchers found that surface dyslexics (subjects who have poor lexical skills, or can’t make out irregular words well) had a mean difference of 14.4 words between reading regular words versus irregular words, however, the mean difference in subjects with phonological dyslexia (subjects who can’t use sub lexical skills, or can’t make out non words) was only 7.75 words which was comparable to the control group . The majority of their subjects showed signs of phonological dyslexia. Twenty-nine subjects showed that their non word reading skills were poorer than their irregular word reading skills. However, sixteen subjects showed the opposite where their irregular word reading skills were poorer than their non word reading skills and were called surface dyslexics .
Surface dyslexia is characterized by subjects who can read non words but who have trouble reading words that are irregular . Surface dyslexia is the outcome of an individual who cannot function using the lexical procedure for reading out loud. The lexical procedure includes sounding out a word though the use of a past word already known . In Castles and Coltheart’s study, the researchers matched 30 regular words with 30 irregular words and asked their surface dyslexic subjects to read the word out loud. As hypothesized in their research, the difference between the surface dyslexic subjects and control subjects was about 4.6 words which was 2.55 words higher than the difference between the phonological dyslexics to the control group . In Castles and Coltheart’s study, both control and dyslexic subjects were shown a card with a word that is irregular or that isn’t pronounced as it looks. Fifteen of the 51 dyslexics were below the confidence limit set by the control subjects on ability to read irregular words. These subjects were then called surface dyslexics .
Phonological dyslexia is characterized by subjects who can read aloud both regular and irregular words but have difficulties with non words and with connecting sounds to symbols, or with sounding out words . Phonological processing tasks predict reading accuracy and comprehension. This subtype is the most predominant form of dyslexia Castles, A., & Coltheart, M. (1993). Varieties of developmental dyslexia. Cognition, 47(2), 149-180 . In Castles and Coltheart’s study, they had 56 dyslexic boys and 56 non dyslexic boys read words and non words given to them. The majority of the boys, 55%, showed a phonological dyslexic pattern .In Castles and Coltheart’s study, dyslexic subjects and control subjects were asked to read non words listed on a card, 17 out of 51 cases of dyslexics were below the confidence limit in non word reading, which was derived by the control group of subjects their own age. These phonological dyslexics have a lower non word reading level than expected by reviewing their irregular word reading level . Phonological dyslexia is the outcome of a subject who cannot function using the sub lexical (pronunciations are constructed from smaller orthographic components) procedure for reading out loud .In Castles and Coltheart’s study, dyslexic and control subjects read words off a note card; the researchers found that while reading irregular words, the dyslexic subjects scored comparable to the control subjects because sub lexical skills weren’t involved in this test .
Double deficit dyslexia
Other researchers have identified a deficit related to “naming speed”, which relates to the ability of students to rapidly verbalize the names of symbols such as letters and numbers when tested . In their study, Wolf and Bowers tested out naming speed by having their subjects name a symbol as quickly as possible when shown on a flash card. Difficulties in naming speed exist in conjunction with a phonological deficit, is characterized as double deficit dyslexia .
The following conditions may also be contributory or overlapping factors, or underlying cause of the dyslexic symptoms as they can lead to difficulty reading:
The following theories should not be viewed as competing, but viewed as theories trying to explain the underlying causes of a similar set of symptoms from a variety of research perspectives and background.
This theory posits that reading is an unnatural act, and carried out by humans for an exceedingly brief period in our evolutionary history (Dalby, 1986). It has been less than a hundred years that most western societies promoted reading by the mass population and therefore the forces that shape our behavior have been weak. Many areas of the world still do not have access to reading for the majority of the population. There is no evidence that "pathology" underlies dyslexia but much evidence for cerebral variation or differences. It is these essential differences that are taxed with the artificial task of reading. The native reading hypothesis of dyslexia is another evolutionary theory which argues that because spoken language is naturally learned in the first few years of development, similarly, written language is best learned at the same early age. It suggests that many forms of dyslexia are therefore, to some extent, a result of introducing reading too late in neurodevelopment. This means that the typically late reading of dyslexics might sometimes be the cause of dyslexia, rather than the other way around, and many cases of dyslexia might be prevented by the earlier introduction of reading instruction.
The phonological hypothesis postulates that dyslexics have a specific impairment in the representation, storage and/or retrieval of speech sounds. It explains dyslexics' reading impairment on the basis that learning to read an alphabetic system requires learning the grapheme/phoneme correspondence, i.e. the correspondence between letters and constituent sounds of speech. If these sounds are poorly represented, stored or retrieved, the learning of grapheme/phoneme correspondences, the foundation of reading by phonic methods for alphabetic systems, will be affected accordingly.
The rapid auditory processing theory is an alternative to the phonological deficit theory, which specifies that the primary deficit lies in the perception of short or rapidly varying sounds. Support for this theory arises from evidence that dyslexics show poor performance on a number of auditory tasks, including frequency discrimination and temporal order judgment. Backward masking tasks, in particular, demonstrate a 100-fold (40 dB) difference in sensitivity between normals and dyslexics. Abnormal neurophysiological responses to various auditory stimuli have also been demonstrated. The failure to correctly represent short sounds and fast transitions would cause further difficulties in particular when such acoustic events are the cues to phonemic contrasts, as in /ba/ versus /da/. There is also evidence that dyslexics may have poorer categorical perception of certain contrasts.
The visual theory (Lovegrove et al., 1980; Livingstone et al., 1991; Stein and Walsh, 1997) reflects another long standing tradition in the study of dyslexia, that of considering it as a visual impairment giving rise to difficulties with the processing of letters and words on a page of text. This may take the form of unstable binocular fixations, poor vergence, or increased visual crowding. The visual theory does not exclude a phonological deficit, but emphasizes a visual contribution to reading problems, at least in some dyslexic individuals. At the biological level, the proposed aetiology of the visual dysfunction is based on the division of the visual system into two distinct pathways that have different roles and properties: the magnocellular and parvocellular pathways. The theory postulates that the magnocellular pathway is selectively disrupted in certain dyslexic individuals, leading to deficiencies in visual processing, and, via the posterior parietal cortex, to abnormal binocular control and visuospatial attention. Evidence for magnocellular dysfunction comes from anatomical studies showing abnormalities of the magnocellular layers of the lateral geniculate nucleus (Livingstone et al., 1991), psychophysical studies showing decreased sensitivity in the magnocellular range, i.e. low spatial frequencies and high temporal frequencies in dyslexics, and brain imaging studies.
Yet another view is represented by the automaticity/ cerebellar theory of dyslexia. Here the biological claim is that the dyslexic's cerebellum is mildly dysfunctional and that a number of cognitive difficulties ensue. First, the cerebellum plays a role in motor control and therefore in speech articulation. It is postulated that retarded or dysfunctional articulation would lead to deficient phonological representations. Secondly, the cerebellum plays a role in the automatization of overlearned tasks, such as driving, typing and reading. A weak capacity to automatize would affect, among other things, the learning of grapheme±phoneme correspondences. Support for the cerebellar theory comes from evidence of poor performance of dyslexics in a large number of motor tasks, in dual tasks demonstrating impaired automatization of balance, and in time estimation, a non-motor cerebellar task. Brain imaging studies have also shown anatomical, metabolic and activation differences in the cerebellum of dyslexics.
There is a unifying theory that attempts to integrate all the findings mentioned above. A generalization of the visual theory, the magnocellular theory postulates that the magnocellular dysfunction is not restricted to the visual pathways but is generalized to all modalities (visual and auditory as well as tactile). Furthermore, as the cerebellum receives massive input from various magnocellular systems in the brain, it is also predicted to be affected by the general magnocellular defect (Stein et al., 2001). Through a single biological cause, this theory therefore manages to account for all known manifestations of dyslexia: visual, auditory, tactile, motor and, consequently, phonological. Beyond the evidence pertaining to each of the theories described previously, evidence specifically relevant to the magnocellular theory includes magnocellular abnormalities in the medial as well as the lateral geniculate nucleus of dyslexics' brains, poor performance of dyslexics in the tactile domain, and the co-occurrence of visual and auditory problems in certain dyslexics.
The concept of a perceptual noise exclusion (Visual-Noise) deficit is an emerging hypothesis, supported by research showing that dyslexic subjects experience difficulty in performing visual tasks such as motion detection in the presence of perceptual distractions, but do not show the same impairment when the distracting factors are removed in an experimental setting. The researchers have analogized their findings concerning visual discrimination tasks to findings in other research related to auditory discrimination tasks. They assert that dyslexic symptoms arise because of an impaired ability to filter out both visual and auditory distractions, and to categorize information so as to distinguish the important sensory data from the irrelevant.
As of 2007, genetic research in families with dyslexia have identified nine chromosome regions that may be associated with susceptibility to dyslexia. However, several of the major studies have not been replicated.
Modern neuroimaging techniques such as functional Magnetic Resonance Imaging (fMRI) and Positron Emission Tomography (PET) have produced clear evidence of structural differences in the brains of children with reading difficulties. It has been found that people with dyslexia have a deficit in parts of the left hemisphere of the brain involved in reading, which includes the inferior frontal gyrus, inferior parietal lobule, and middle and ventral temporal cortex.
Scientific studies of brains donated to medical research have revealed that there are anatomical differences in two parts of the dyslexic brain: the cerebral cortex and the thalamus. In 1979 Albert Galaburda of Harvard Medical School noticed anatomical differences in the language center in a dyslexic brain, showing microscopic differences known as ectopias and microgyria. Both affect the typical six-layer structure of the cortex. An ectopia is a collection of neurons that have pushed up from the lower layers of the cortex into the outermost one. A microgyrus is an area of cortex that includes only four layers instead of six. These differences affect connectivity and functionality of the cortex in critical areas related to auditory processing and visual processing, which seems consistent with the hypothesis that dyslexia stems from a phonological awareness deficit.
Studies of both autopsied brains and living brains using neuroimaging techniques have shown that the brains of dyslexic children are symmetrical, unlike the asymmetrical brains of non-dyslexic readers who had larger left hemispheres.
Scientists do not claim that all people with dyslexia have these structural brain differences. However, the studies are evidence that some children's reading problems are brain based. The challenge for researchers is to determine how these structural differences affect reading acquisition.
Some studies have concluded that speakers of languages whose orthography has a highly consistent correspondence between letter and sound (e.g., Italian) suffer less from the effects of dyslexia than speakers of languages where the letter-sound correspondence is less consistent (e.g. English and French).
In one of these studies, reported in Seymour et al., the word-reading accuracy of first-grade children of different European languages was measured. English children had an accuracy of just 40%, whereas among children of most other European languages accuracy was about 95%, with French and Danish children somewhere in the middle at about 75%; Danish and French are known to have an irregular pronunciation.
However, this does not mean that dyslexia is caused by orthography: instead, Ziegler et al. claim that the dyslexia suffered by German or Italian dyslexics is of the same kind as the one suffered by the English ones, supporting the theory that the origin of dyslexia is biological. In a study by Paulescu et al. (Science, 2001) English, French, and Italian dyslexics were found to have the same brain function signature when studied with functional magnetic resonance imaging (fMRI), a signature that differed from non-dyslexic readers. However, dyslexia has more pronounced effects on orthographically difficult languages, e.g., dyslexics have more difficulty in English than Italian. Modern theories of some forms of dyslexia uses orthography to test a hypothesis of psychological causation
Recent advances in neuroimaging and genetics provide evidence that could potentially help identify children with dyslexia before they learn to read in the future. However, such tests have not yet been developed and more research is needed before such testing could be considered reliable.
Speech delays may be an early warning sign of dyslexia. Many dyslexics may have problems processing and decoding auditory input prior to reproducing their own version of speech. Early stuttering or cluttering can also be warning signs of dyslexia.
Many dyslexics also can have problems with speaking clearly. They can mix up sounds in multi-syllabic words (ex: aminal for animal, bisghetti for spaghetti, hekalopter for helicopter, hangaberg for hamburger, mazageen for magazine, etc.) They also can have problems speaking in full sentences. They can have trouble correctly articulating Rs and Ls as well as Ms and Ns. They often have "immature" speech. They may still be saying "wed and gween" instead of "red and green" in second or third grade. Many dyslexics might have speech therapy in special education. They may have fast speech, cluttered speech, or hesitant speech.
Reading requires the sounding out of words. Therefore, it makes sense that children with speech problems can end up having reading problems later. Many have problems with speech due to problems with auditory processing disorder issues.
Many dyslexics have problems with phonemic awareness. Phonemes are the smallest units in spoken language. The Auditory related underlying causes of dyslexia may be partially remediated by auditory therapy or speech therapy, which help with phonemic awareness. This may help to make sense of phonics which may help with phonological awareness, which is needed to sound out words.
Some shared symptoms of the speech/hearing deficits and dyslexia:
Because of literacy problems, an individual with dyslexia may have difficulty with handwriting. This can involve slower writing speed than average or poor handwriting characterised by irregularly formed letters. They may use inappropriate words when writing.
Some studies have also reported gross motor difficulties in dyslexia, including motor skills disorder. This difficulty is indicated by clumsiness and poor coordination. The relationship between motor skills and reading difficulties is poorly understood but could be linked to the role of the cerebellum and inner ear in the development of reading and motor abilities.
Dyslexia should not be confused with dyscalculia, a learning disability marked by severe difficulties with mathematics. Individuals with dyslexia can be gifted in mathematics while having poor reading skills. However, in spite of this they might have difficulty with word problems (i.e., descriptive mathematics, engineering, or physics problems that rely on written text rather than numbers or formulas). Individuals with dyslexia may also have difficulty remembering mathematical facts, such as multiplication tables, learning the sequence of steps when performing calculations, such as long division, and other mathematics which involve remembering the order in which numbers appear. This may be exhibited by having a slow response in mathematical drills and difficulty with word problems.
A study has found that entrepreneurs are five times more likely to suffer from dyslexia than average citizens.
There is no cure for dyslexia, but dyslexic individuals can learn to read and write with appropriate education or treatment. There is wide research evidence indicating that specialized phonics instruction can help remediate the reading deficits. The fundamental aim is to make children aware of correspondences between graphemes and phonemes, and to relate these to reading and spelling. It has been found that training, that is also focused towards visual language and orthographic issues, yields longer-lasting gains than mere oral phonological training.
Teachers are also using audiobooks as a way of teaching textbooks in an engaging way to those with dyslexia. In the UK, one of the biggest charities is Listening Books, offering members a streaming service over the internet. www.listening-books.org.uk An Australian company, ReadHowYouWant is working to make all published books available in audiobook form.
Accessible publishing, the method by which charities and companies work to provide books and textbooks in a variety of formats and fonts suitible for all readers, has long been focused on developing formats to improve dyslexia by use of word patterns, phonic symbols, highlighting mirrored letters (such as b and d), and increasing the font size as words move along.
Effective training is given by teachers at school or kindergarten. Meta-analysis evaluating the effects of phonological awareness instruction has shown that word reading skills of all children, those with a risk for reading problems as well as those developing typically, improved their reading in systematic phonics instruction, a method that encourages a word to be recognised through the building of its constituent sounds. Basic phonemic awareness instruction did not, however, improve spelling in disabled readers. None of the studies included measures of reading fluency.
The core deficit of dyslexia is in learning to read at the word level , and individuals with dyslexia require more practice to master skills in their areas of deficit. In the circumstances where typically developing children need 30 to 60 hours training, the number of hours that has resulted in optimistic conclusions concerning the treatability of dyslexia is between 80 and 100 hours, or less if the intervention is started sufficiently early. Only approximately 20% of adults with early reading difficulties have acquired fluent reading skills in adulthood.
Functional MRI (fMRI) studies have shown changes in the brains of dyslexic children and adults with phonics tutoring, along with improved performance on tests of phonemic awareness and text decoding. Functional MRI studies have also shown changes in the brain and spelling improvement of dyslexic children taught spelling phonetically in an orthographic manner.
One factor that characterises the field of dyslexia treatment is the incessant flow of alternative therapies for developmental and learning disabilities. These controversial treatments include nutritional supplements, special diets, homeopathy, and osteopathy/chiropractic manipulation.
Dyslexia's main manifestation is a difficulty in developing word-level reading skills in elementary school children. Those difficulties result from reduced ability to associate visual symbols with verbal sounds. While motivational factors must also be reviewed in assessing poor performance, dyslexia is considered to be developmental. Most scientific criteria for dyslexia exclude cases that can be explained as arising from environmental factors such as lack of education or total sensory deficits.
Dyslexia can be substantially compensated for with proper therapy, training, and assistive technology. Many coping strategies are developed subconsciously by the individual dyslexic.
Dyslexia has many variations dependent on the cultural choice of visual notation of speech. So the nature of the notation used in different cultures creates different types of problems for their groups of dyslexics. The differences between the English text and Chinese characters is a good example.
Some charitable organizations like the Scottish Rite Foundation have undertaken the task of testing for dyslexia and making training classes and materials available, often without cost, for teachers and students.
In England and Wales, the failure of schools to diagnose and provide remedial can help for dyslexia following the House of Lords decision in the case of Pamela Phelps has created an entitlement for students with dyslexia in Higher education to receive support funded via the Disabled Students Allowance. Support can take the form of IT equipment (software and hardware) as well as personal assistance, also known as non-medical helper support. Dyslexic students will also be entitled to special provision in examinations such as additional time to allow them to read and comprehend exam questions.
The British Disability Discrimination Act 1995 also covers dyslexia.
Some disagreement exists as to whether dyslexia does indeed exist as a condition, or whether it simply reflects individual differences among different readers.
"The Dyslexia Myth" is a documentary that appeared as part of the Dispatches series produced by British broadcaster Channel 4. First aired in September 2005, it claims to expose myths and misconceptions that surround dyslexia. It argues that the common understanding of dyslexia is not only false but makes it more difficult to provide the reading help that hundreds of thousands of children desperately need. Drawing on years of intensive academic research on both sides of the Atlantic, it challenged the existence of dyslexia as a separate condition, and highlighted the many different forms of reading style.
The documentary only focused on the reading difficulties that dyslexics encounter. As discussed in previous headings, dyslexia is more than a mere reading disability, and commonly includes symptoms that extend beyond reading difficulties. However, these symptoms are not included in the DSM-IV list of symptoms by which "Reading Disorder" is diagnosed (USA).
Julian Elliott, a psychologist at Durham University in the UK, disputes the characterization of dyslexia as a medical condition, and believes it should be treated simply as a reading difficulty. According to Elliot, "[parents] don’t want their child to be considered lazy, thick or stupid. If they get called this medically diagnosed term, dyslexic, then it is a signal to all that it’s not to do with intelligence.” Elliot believes that children of all levels of intelligence may struggle with learning to read, and that all can be helped by educational strategies appropriate to their needs. He feels that resources are wasted on diagnosis and testing, and favors early intervention programs for all struggling readers.
However, John Everatt of the University of Surrey, also in the UK, has suggested that dyslexic students can be distinguished from other children with low reading achievement by testing geared to assessing their strengths as well as weaknesses. Dyslexic children tend to score significantly better than other children, including non-impaired children, on tests of creativity, spatial memory, and spatial reasoning. Dyslexic children also perform better than other reading-impaired children on tests of vocabulary and listening comprehension. Everatt suggests that dyslexic children may be better served by educational intervention which includes strategies geared to their unique strengths in addition to skill remediation, and thus recommends more comprehensive evaluation and targeted interventions.
Dyslexic characters have featured in a number of works of fiction. Notable works include Aamir Khan's 2007 award - winning film Taare Zameen Par, Henry Winkler's Hank Zipzer series of children's books, and Jennifer Weiner's 2002 novel, In Her Shoes (which was adapted as the 2005 film, In Her Shoes).
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